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Preclinical analysis demonstrates that 1-methyl-D-tryptophan enhances CD8+ T cell-macrophage interactions in tuberculosis granulomas, indicating potential for host-directed therapies.
T cell infiltration and reduces T cell activation and proliferation; and iii) therapeutic strategies, including D-1MT, that improve intragranulomatous killing hold strong translational potential.
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McCaffrey et al. (2026) studied this question.
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