Key result
Inhibition of K(v)7 channels with linopirdine, XE991, or KCNQ3 morpholinos induced burst discharge activity and convulsive behaviors in zebrafish larvae, which was blocked by retigabine.
Functional K(v)7 channels are expressed in the larval zebrafish nervous system and play a direct role in the generation of seizure activity, establishing a model for studying neurological disorders like benign familial neonatal convulsion.
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Zebrafish model validates K(v)7 role in seizures; leaves open translation to human epilepsy therapies.
Members of the K(v)7 family generate a subthreshold potassium current, termed M-current, that regulates the excitability of principal central neurons. Mutations in two members of this family, K(v)7.2 (KCNQ2) and K(v)7.3 (KCNQ3) are associated with a neurological disorder known as benign familial neonatal convulsion (BFNC). Despite their importance in normal and pathological brain function, developmental expression and function of these channels remains relatively unexplored. Here, we examined the temporal expression of K(v)7 channel subunits in zebrafish larvae using a real-time quantitative PCR approach. Spatial expression in the larval zebrafish brain was assessed using whole-mount in situ hybridization. The mRNA for three members of the K(v)7 family (KCNQ2, 3 and 5) is reported in zebrafish between two and seven days post-fertilization (dpf). Using electrophysiological techniques, we show that inhibitors of K(v)7 channels (linopirdine and XE991) induce burst discharge activity in immature zebrafish between 3 and 7 dpf. This abnormal electrical activity is blocked by a K(v)7 channel opener (retigabine) and was also shown to evoke convulsive behaviors in freely swimming zebrafish. Using morpholino oligonucleotides directed against KCNQ3, we confirmed a role for KCNQ channels in generation of electrical burst discharges. These results indicate that functional K(v)7 channels are expressed in the larval zebrafish nervous system and could play a direct role in generation of seizure activity.
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Chege et al. (2011) studied Seizure activity / Benign familial neonatal convulsion. K(v)7 channel inhibitors (linopirdine and XE991) and KCNQ3 morpholino oligonucleotides was evaluated on Burst discharge activity and convulsive behaviors. Inhibition of K(v)7 channels with linopirdine, XE991, or KCNQ3 morpholinos induced burst discharge activity and convulsive behaviors in zebrafish larvae, which was blocked by retigabine.
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