Acute myocardial infarction is associated with transient shortening of the QS2 interval and ejection time, independent of urinary catecholamine excretion levels.
Post-MI shortening of QS2 interval and ejection time appears independent of catecholamines; hypothesis-generating for alternative mechanisms without supporting practice changes.
After acute myocardial infarction there is a shortening of the QS2 interval and the ejection time. The pre-ejection time is either normal or prolonged. After the stage of maximum shortening of the QS2 interval and ejection time, there is a progressive improvement towards the normal. In this study there was no correlation between the shortened QS2 interval and ejection time, and the level of urinary catecholamine excretion. The physiological and clinical significance of these changes in the systolic time intervals and their relation to the altered haemodynamics and mechanics are discussed.
No takes yet. Share an insight, caveat, or question.
Roland Samson (1970) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: