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November 20, 2007FEBS LettersOpen Access

Diosgenin, a naturally occurring steroid, suppresses fatty acid synthase expression in HER2‐overexpressing breast cancer cells through modulating Akt, mTOR and JNK phosphorylation

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Authors

CCChun-Te ChiangLarry Ellison FoundationTWTzong‐Der WayChina Medical UniversitySTShang-Jie Tsai

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Cite This Study

Chiang et al. (2007) studied this question.

synapsesocial.com/papers/6a86f16e36c36f97809ec645https://doi.org/10.1016/j.febslet.2007.11.021
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Activation of the Akt/Mammalian Target of Rapamycin/4E-BP1 Pathway by ErbB2 Overexpression Predicts Tumor Progression in Breast Cancers2004 · 313 citations
  2. 2Studies of the HER-2/ <i>neu</i> Proto-Oncogene in Human Breast and Ovarian Cancer1989 · 6,868 citations
  3. 3Fatty acid synthase, a proficient multifunctional enzyme1989 · 822 citations
  4. 4Role of MAPKs and NF-κB in diosgenin-induced megakaryocytic differentiation and subsequent apoptosis in HEL cells2006 · 53 citations