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June 1, 1996Infection and ImmunityOpen Access

Leishmania major-human macrophage interactions: cooperation between Mac-1 (CD11b/CD18) and complement receptor type 1 (CD35) in promastigote adhesion

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Authors

LRL. A. RosenthalUniversity of Wisconsin–MadisonFSFayyaz S. SutterwalaCedars-Sinai Medical CenterMKMarcus E. KehrliAgricultural Research Service

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Cite This Study

Rosenthal et al. (1996) studied this question.

synapsesocial.com/papers/6a870ad4540b033b21bf06e1https://doi.org/10.1128/iai.64.6.2206-2215.1996
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Evidence for distinct intracellular pools of receptors for C3b and C3bi in human neutrophils.1985 · 289 citations
  2. 2CR1, the C3b receptor, mediates binding of infective Leishmania major metacyclic promastigotes to human macrophages.1989 · 257 citations
  3. 3Molecular definition of the bovine granulocytopathy syndrome: Identification of deficiency of the Mac-1 (CD11b/CD18) glycoprotein1990 · 192 citations
  4. 4Role of the Leishmania surface protease gp63 in complement fixation, cell adhesion, and resistance to complement-mediated lysis1995 · 327 citations
  5. 5The mouse macrophage receptor for C3bi (CR3) is a major mechanism in the phagocytosis of Leishmania promastigotes.1985 · 263 citations