Why the study?
Trypanosomatids present significant structural differences in their 40S compared to mammalian hosts, suggesting substantial variability in translation initiation.
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Design
Structural biology study with GST-pulldown assays and mass spectrometry
Structural differences in trypanosomatid 43S PIC assembly; leaves open selective targeting for parasitic diseases.
SUMMARY Canonical mRNA translation in eukaryotes begins with the formation of the 43S pre-initiation complex (PIC). Its assembly requires the binding of several eukaryotic initiation factors (eIF 1, 1A, 2, 3 and 5), Met-tRNA i Met and the small ribosomal subunit (40S). Compared to their mammalian hosts, trypanosomatids present significant structural differences in their 40S suggesting substantial variability in translation initiation. Here we determined the structure of the 43S PIC from Trypanosoma cruzi , the parasite causing the Chagas disease. Our structure shows numerous specific features, such as the variant eIF3 structure and its unique interactions with the large rRNA ESs 9 S , 7 S and 6 S , and the association of a kinetoplastid-specific ~245 kDa DDX60-like helicase. It also revealed the so-far-elusive 40S-binding site of the eIF5 C-terminal domain and the structures of key terminal tails of several conserved eIFs underlying their activities within the PIC. Our results are corroborated by GST-pulldown assays in both human and T. cruzi and mass-spectrometry data.
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Bochler et al. (2019) studied this question.
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