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The ability of furosemide to increase RBF was found to be significantly inhibited by indomethacin. The implication of this observation is that the dilation of the renal vasculature produced by furosemide is mediated by prostaglandins. Inhibition of the synthesis of these agents by indomethacin inhibits the increase in RBF produced by furosemide.
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Williamson et al. (1975) studied this question.
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