The use of repair deficient strains of bacteria for detecting mutagenic properties of chemical species is now an established technique. In this paper we present inactivation results obtained with Escherichia coli K12 AB2480 ( uvr A rec A ) which indicate that p ‐aminobenzoic acid, a common component of sunscreening formulations, may increase the frequency of lethal damage induced in DNA when cell populations are exposed to near ultraviolet radiation. Preliminary experiments, utilising the selective action of photoenzymatic repair, indicated that the sensitisation to near ultraviolet radiation is partially but not wholly due to increased formation of pyrimidine dimers.
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Hodges et al. (1977) studied this question.
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