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May 15, 1994The Journal of PhysiologyOpen Access

ATP released together with acetylcholine as the mediator of neuromuscular depression at frog motor nerve endings.

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Authors

RRR.S. RedmanESEugene M. SilinskyNorthwestern University

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Redman et al. (1994) studied this question.

synapsesocial.com/papers/6a884166d79ae02bd34e0090https://doi.org/10.1113/jphysiol.1994.sp020176
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1On the role, inactivation and origin of endogenous adenosine at the frog neuromuscular junction.1987 · 146 citations
  2. 2Calcium currents at motor nerve endings: absence of effects of adenosine receptor agonists in the frog.1992 · 65 citations
  3. 3Estimates of probability of transmitter release at the mammalian neuromuscular junction1970 · 122 citations
  4. 4Synaptic vesicle exocytosis captured by quick freezing and correlated with quantal transmitter release.1979 · 1,424 citations
  5. 5EVIDENCE FOR SPECIFIC ADENOSINE RECEPTORS AT CHOLINERGIC NERVE ENDINGS1980 · 74 citations