Why the study?
Does continuous administration of AMT prevent postischemic heart dysfunction and injuries in stroke-prone spontaneously hypertensive rats?
Does continuous administration of AMT prevent postischemic heart dysfunction and injuries in stroke-prone spontaneously hypertensive rats?
Inhibition of iNOS with AMT ameliorates postischemic cardiac dysfunction and injury in a hypertensive and hypertrophic rat model.
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iNOS inhibition with AMT may reduce postischemic injury in hypertensive rat models; leaves open translation to human ischemia or HF.
Abe et al. (2001) studied this question.
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