Why the study?
Is stated alcohol consumption associated with increased blood pressure in working adults not on antihypertensive treatment?
Is stated alcohol consumption associated with increased blood pressure in working adults not on antihypertensive treatment?
Alcohol consumption has a linear, independent association with increased systolic and diastolic blood pressure in adults not on antihypertensive therapy, with no apparent threshold effect.
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May inform alcohol assessment during BP evaluation; leaves open causality and intervention effects for prospective trials.
We studied the association between stated alcohol consumption and blood pressure, making allowance for age, adiposity and smoking in 13 535 men and 7385 women who were not receiving antihypertensive treatment. They represented a wide cross-section of the inner Sydney working population with 95% aged between 18 and 70. We found a high degree of linear correlation between stated alcohol consumption and blood pressure, diastolic and systolic. This relationship was independent of age, adiposity and smoking. For each 100 g/week increase in stated alcohol consumption, diastolic blood pressure increased by 0.12 kPa (0.92 mmHg) in men and by 0.20 kPa (1.5 mmHg) in women; no threshold for this effect was evident. A plateau appeared at about 500 g/week. Blood pressure increased significantly with age and adiposity (Quetelet's index). Smoking was associated with a lower diastolic blood pressure. The difference in mean diastolic blood pressure between smokers and non-smokers was 0.20 kPa (1.5 mmHg) for men and 0.27 kPa (2.1 mmHg) for women.
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Cooke et al. (1982) studied this question.
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