Why the study?
The mechanisms of rheumatic diseases remain ill-defined and targeted therapies challenging, while cell metabolism plays important roles in their pathogenesis.
This review highlights the role of lipid metabolism profiles, including dyslipidaemia and fatty acid metabolism, in the pathogenesis of rheumatic diseases.
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Supports expanded research on lipid targets in rheumatic diseases; leaves open whether modulation alters clinical outcomes.
Rheumatic diseases are a group of chronic autoimmune disorders that involve multiple organs or systems and have high mortality. The mechanisms of these diseases are still ill-defined, and targeted therapeutic strategies are still challenging for physicians. Recent research indicates that cell metabolism plays important roles in the pathogenesis of rheumatic diseases. In this review, we mainly focus on lipid metabolism profiles (dyslipidaemia, fatty acid metabolism) and mechanisms in rheumatic diseases and discuss potential clinical applications based on lipid metabolism profiles.
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Chen et al. (2021) studied this question.
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