Why the study?
The exact pathogenesis of shoulder joint contractures and glenohumeral joint dysplasia in obstetrical brachial plexus palsy remains unknown, and understanding it is needed to develop effective prophylactic treatment.
This review summarizes the current theories on the pathogenesis of glenohumeral deformity and contractures following obstetric brachial plexus palsy to aid in developing prophylactic treatments.
May guide prophylactic treatment development in obstetric brachial plexus palsy; leaves open validation of denervation-induced growth hypotheses.
Contractures of the shoulder joint and glenohumeral joint dysplasia are well known complications to obstetrical brachial plexus palsy. Despite extensive description of these sequelae, the exact pathogenesis remains unknown. The prevailing theory to explain the contractures and glenohumeral joint dysplasia states that upper trunk injury leads to nonuniform muscle recovery and thus imbalance between internal and external rotators of the shoulder. More recently, another explanation has been proposed, hypothesizing that denervation leads to reduced growth of developing muscles and that reinnervation might suppress contracture formation. An understanding of the pathogenesis is desirable for development of effective prophylactic treatment. This article aims to describe the current state of knowledge regarding these important complications.
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Olofsson et al. (2019) studied this question.
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