Why the study?
Myocardial fibrosis is a hallmark pathological outcome of many cardiovascular diseases and a key component of cardiac remodeling, with MAPK signaling pathways being central to this process.
Design
Review
Authors
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Identifies MAPK targets for myocardial fibrosis therapy; leaves open clinical translation pending prospective trials.
The p38 MAPK, JNK, and ERK signaling pathways play a central role in myocardial fibrosis and represent potential therapeutic targets.
Wu et al. (2025) studied this question.
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