⌘+K

Pulse Explore Clubs Researchers Journals

Home Clubs Explore

September 15, 2002Journal of NeuroscienceOpen Access

Calmodulin Is an Auxiliary Subunit of KCNQ2/3 Potassium Channels

View Full Paper

Ask AI

Bookmark

Share

Authors

HWHua WenOregon Health & Science University ILIrwin B. LevitanThomas Jefferson University

Discussion

Loading...

Member takes

Implication

May suggest KCNQ targets in epilepsy; leaves open in vivo functional validation.

Key Points

Key points are not available for this paper at this time.

Cite This Study

Wen et al. (2002) studied this question.

synapsesocial.com/papers/6a896eeb40fe6a3effcb0895 https://doi.org/10.1523/jneurosci.22-18-07991.2002

View Full Paper

Ask AI

Bookmark

Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Calmodulin Regulates Assembly and Trafficking of SK4/IK1 Ca2+-activated K+ Channels2001 · 126 citations
2Differential Expression of KCNQ2 Splice Variants: Implications to M Current Function during Neuronal Development2001 · 76 citations
3Functional Expression of Two KvLQT1-related Potassium Channels Responsible for an Inherited Idiopathic Epilepsy1998 · 200 citations
4Requirement of a Macromolecular Signaling Complex for β Adrenergic Receptor Modulation of the KCNQ1-KCNE1 Potassium Channel2002 · 720 citations
5Regulation of RYR1 Activity by Ca<sup>2+</sup> and Calmodulin2000 · 117 citations