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AIM: exonic mutations in these models as well as in diverse types of human cancer samples including AMLs. METHODS: was further assessed in diverse types of cancer samples including AMLs in the Cancer Genome Atlas (TCGA) at the National Cancer Institute. RESULTS: also exhibited a very low mutational frequency in AMLs compared with all types of human cancer. In addition, we found that CsA played a role in undermining the selection of highly drug-resistant cells via induction of low-level and unstable drug resistance. CONCLUSION: mutations.
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Chen et al. (2020) studied this question.
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