Key result
OCT-guided percutaneous coronary intervention successfully treated a STEMI caused by spontaneous coronary artery dissection, with histology revealing unique intimal inflammatory infiltrates.
Case Report (n=1)
No
OCT is a valuable tool for guiding PCI in STEMI secondary to SCAD, and in vivo histology suggests that intimal inflammation may contribute to the etiology of SCAD.
Spontaneous coronary artery dissection (SCAD) is a well-known but infrequent cause of acute coronary syndrome (ACS), and often goes unrecognized. Although management of SCAD is, at times, controversial, when a patient presents with ACS, percutaneous coronary intervention (PCI) is frequently necessary. We present a patient with ST-segment elevation myocardial infarction (STEMI) with SCAD that illustrates two important points: use of intracoronary optical coherence tomography to guide PCI, and histologic assessment to provide a unique insight into the etiology of SCAD. Following the case, we briefly review the important aspects of the pathophysiology, epidemiology, diagnosis, and interventional management of SCAD.
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Krishnan et al. (2017) conducted a case report in Spontaneous Coronary Artery Dissection (SCAD) (n=1). Optical coherence tomography (OCT) guided percutaneous coronary intervention (PCI) was evaluated on Clinical and angiographic success. OCT-guided percutaneous coronary intervention successfully treated a STEMI caused by spontaneous coronary artery dissection, with histology revealing unique intimal inflammatory infiltrates.
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