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November 26, 2003Annals of Neurology

Ribonuclear inclusions in skeletal muscle in myotonic dystrophy types 1 and 2

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Authors

AMAmi MankodiNational Institute of Neurological Disorders and Stroke PTPatana Teng-umnuayDhurakij Pundit University MKMatt KrymUniversity of Rochester

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Implication

Muscleblind sequestration may contribute to DM muscle pathology; leaves open its role relative to disease severity and PKR independence.

Key Points

Key points are not available for this paper at this time.

Cite This Study

Mankodi et al. (2003) studied this question.

synapsesocial.com/papers/6a89a6904c30e1095aec84b2 https://doi.org/10.1002/ana.10763

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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Cardiac elav-type RNA-binding protein (ETR-3) binds to RNA CUG repeats expanded in myotonic dystrophy1999 · 116 citations
2RNA editing activity is associated with splicing factors in lnRNP particles: The nuclear pre-mRNA processing machinery2001 · 88 citations
3Glutamine Repeats and Neurodegeneration2000 · 1,322 citations
4Somatic instability of CTG repeat in myotonic dystrophy1993 · 198 citations
5Histopathological differences of myotonic dystrophy type 1 (DM1) and PROMM/DM22003 · 173 citations