TAE alone may not induce ischemia-induced tumor necrosis in ruptured hepatic angiosarcoma, suggesting it might be unsuitable as a definitive treatment.
TAE may not induce tumor necrosis in hepatic angiosarcoma; leaves open optimal hemostasis strategies for this rare tumor.
An 80-year-old Japanese man presented to our hospital with intra-abdominal hemorrhage due to a ruptured liver tumor. Transcatheter arterial embolization (TAE) temporarily achieved hemostasis, but he died following re-rupture 4 days later. Based on autopsy findings, the liver tumor was diagnosed as hepatic angiosarcoma. Embolic agents used during embolization were identified within the hepatic small interlobular arteries. However, there were no findings of tumor cell necrosis or ischemic change in the angiosarcoma. In the present case, TAE alone did not induce ischemia-induced tumor necrosis, suggesting that TAE might be unsuitable to treat hepatic angiosarcoma. Treatment optimization for ruptured hepatic angiosarcoma is desired.
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Entani et al. (2023) studied this question.
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