Population
Atherosclerosis-prone apolipoprotein E-deficient mice; 2,000 patients with coronary artery disease; and 782…
Comparison
MMP8 gene inactivation and MMP8 gene variation vs MMP8+/+/apoE-/- controls and alternative alleles
Design
Preclinical
Follow-up
12 weeks (mice); 10 years (Bruneck study)
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Supports MMP8 inhibition for atherosclerosis testing; leaves open translation to human event reduction.
MMP8 plays a causal role in atherogenesis, and its genetic variation is associated with the extent and progression of atherosclerosis in humans.
A 2009 study studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: