Why the study?
The relation between myocardial and vascular injury in the generation of the no-reflow phenomenon remained to be explored.
Does the degree of no-reflow correlate with the extent of infarction during maximal vasodilation in a porcine model of ischemia-reperfusion?
Does the degree of no-reflow correlate with the extent of infarction during maximal vasodilation in a porcine model of ischemia-reperfusion?
In a porcine model, the degree of no-reflow correlates closely with the extent of infarction during maximal vasodilation, and this decrease in vascular conductance is not prevented by aspirin.
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Suggests no-reflow as infarct surrogate in porcine models; hypothesis-generating and should not yet inform clinical practice.
Johnson et al. (1988) studied this question.
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