Commentary highlights cyclic alternating pattern dynamics in sleep apnea patients with elevated intracranial pressure, suggesting a neglected neural mechanism in nocturnal pressure spikes.
recently published an interesting paper on the association between intracranial pressure (ICP) elevation, measured through the Lundberg B waves, and sleep apnea in a group of patients with idiopathic intracranial hypertension (IIH) and hydrocephalus [1].ICP B waves are defined as short, repetitive elevation of intracranial pressure of up to 50 mmHg with a frequency of 0.5-2 waves/min, which are typically observed in patients with IIH, but can also be measured in subjects with normal intracranial pressure [2].Obstructive sleep apnea (OSA) is a multi-systemic syndrome characterized by phasic interruptions of airflow during sleep, leading to severe sleep fragmentation and cardiovascular consequences, presenting a typical 20-40 s periodicity (Panel A, Fig. 1).According to Riedel et al.[1], there is an interesting association between ICP B waves and sleep apnea.The
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Mutti et al. (2024) studied this question.
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