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SUMMARYTwelve normal subjects, 7 of whom showed slowing of cerebral rhythms during hyperventilation, were semipassively hyperventilated at high respiratory volumes while EEG, ERG, and expiratory CO2 were being recorded. Eleven subjects showed some increase of the ERG b-wave amplitude, but those with the greatest increase also had the most “slowing” of EEG frequencies. The prolonged ERG b-wave amplitude increase is evidence against the presence of HV-induced hypoxia in the retina and suggests that slowing of cerebral rhythms in HV is also due to factors other than hypoxia.
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Morgan et al. (1970) studied this question.
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