Why the study?
It remained unknown whether AT4Rs act alone or with AT2Rs in mediating the cognitive and cerebrovascular benefits of angiotensin receptor blockers in Alzheimer disease models.
Does AT2R modulation improve cognitive and cerebrovascular deficits in a mouse model of Alzheimer disease?
Population
Transgenic mice overexpressing familial Alzheimer disease mutations of the human APP
Comparison
Losartan plus PD123319 vs losartan plus vehicle, and compound 21 vs vehicle
Design
Preclinical animal study
Authors
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AT2R modulation fails to restore cognition or neurovascular function in AD mice; leaves open any therapeutic role in human disease.
Does AT2R modulation improve cognitive and cerebrovascular deficits in a mouse model of Alzheimer disease?
Targeting AT2Rs does not appear to be an effective strategy for restoring Aβ-related cognitive and cerebrovascular deficits in Alzheimer's disease models.
Royea et al. (2020) studied this question.
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