Key result
Ankylosing spondylitis was associated with prolonged atrial electromechanical delay and increased epicardial adipose tissue thickness, which correlated with interatrial EMD (r=0.451, p<0.001).
Why the study?
To evaluate whether atrial electromechanical delay and epicardial adipose tissue thickness differed between patients with ankylosing spondylitis and healthy subjects.
Are atrial electromechanical delay and epicardial adipose tissue thickness increased in patients with ankylosing spondylitis compared to healthy subjects?
Cross-Sectional (n=85)
No
Are atrial electromechanical delay and epicardial adipose tissue thickness increased in patients with ankylosing spondylitis compared to healthy subjects?
Ankylosing spondylitis is associated with prolonged atrial electromechanical delay and increased epicardial adipose tissue, suggesting subclinical arrhythmogenic and cardiovascular involvement linked to inflammation.
Suggests subclinical arrhythmogenic risk in ankylosing spondylitis.
In this study, we aimed to evaluate whether atrial electromechanical delay (EMD) and epicardial adipose tissue (EAT) thickness differed between ankylosing spondylitis (AS) patients and healthy subjects. Methods: This prospective, cross-sectional study included 43 consecutive AS patients followed up in the Physical Medicine and Rehabilitation Department of the University of Health Sciences Turkey, stanbul Training and Research Hospital, between June 2019 and January 2020. The control group consisted of 42 age-and gender-matched healthy participants. The PA atrial EMD was accepted as the beginning of the P wave on the electrocardiograph and the begining of late diastolic wave (Am wave) on the tissue Doppler obtained by transthoracic echocardiography, and all EMD parameters, including lateral mitral annulus (lateral PA), septal mitral annulus (septal PA) and right ventricular tricuspid annulus (tricuspid PA), were calculated. The thickness of EAT was obtained from the thickest part of the right ventricular free wall at the end of diastole in the parasternal long axis window. Results: In AS patients, tissue Doppler measurements of PA lateral, PA septal and PA tricuspid were longer than the measurements in the control group. In addition, EAT thickness was significantly higher in AS patients than in the control group. There was a moderate correlation between interatrial EMD and C-reactive protein (r=0.445, p<0.001) and EAT thickness (r=0.451, p<0.001).
No takes yet. Share an insight, caveat, or question.
Öz et al. (2020) conducted a cross-sectional in Ankylosing Spondylitis (n=85). Ankylosing spondylitis vs. Healthy controls was evaluated on Atrial electromechanical delay (EMD) and epicardial adipose tissue (EAT) thickness. Ankylosing spondylitis was associated with prolonged atrial electromechanical delay and increased epicardial adipose tissue thickness, which correlated with interatrial EMD (r=0.451, p<0.001).
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: