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Case report and review reveals traumatic tricuspid regurgitation causing refractory shock after blunt chest trauma, highlighting the critical role of early structural cardiac evaluation.
In polytrauma patients, persistent or unexplained hemodynamic instability is commonly attributed to hemorrhage, hypovolemia, or septic evolution. Compensatory neuro-cardiovascular responses may temporarily mask structural cardiac injury and delay diagnosis. Traumatic tricuspid valve injury is a rare condition in which physiological compensation and right-ventricular adaptation can initially preserve systemic perfusion but progressively impair circulatory efficiency. We report the case of a young polytrauma patient and a targeted narrative review to shed light on a rare cause of hemodynamic instability in blunt chest trauma, emphasizing that identifying the underlying mechanism of shock is crucial for adequate management.
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Angeletti et al. (2026) studied this question.
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