EVIDENCE will be presented in this communication to support the hypothesis that anisometropia per se does not produce strabismus, but that, for hypermetropes, the refraction of the better eye is an important factor. Further, the clinical entity of anisometropic amblyopia and its treatment will be discussed. Finally, some comments will be made on optical correction in anisometropia. The case records used for the statistical analysis were all obtained from Moorfields Eye Hospital (High Holborn Branch); others came from a variety of sources, including Bristol Eye Hospital.
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C. I. Phillips (1959) studied this question.
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