Why the study?
Does biatrial anastomosis heart transplantation increase atrial electromechanical delay compared to healthy controls?
Does biatrial anastomosis heart transplantation increase atrial electromechanical delay compared to healthy controls?
Patients who underwent biatrial anastomosis heart transplantation have prolonged inter- and intra-atrial electromechanical delay, which may explain their increased incidence of atrial arrhythmias.
Background We aimed to assess atrial electromechanical delay (AEMD) in patients who had undergone heart transplantation. Methods A total of 32 patients who underwent biatrial anastomosis heart transplantation (24 men, 8 women; mean age: 42±11 years) and 30 healthy volunteers (20 men, 10 women; mean age: 36±13 years) were included in the study. Atrial electromechanical coupling (PA), intra‐AEMD, and inter‐AEMD were measured. Results PA lateral (68±7 vs. 51±11 ms, p<0.01), PA septal (50±5 vs. 42±8 ms, p< 0.01) and PA tricuspid (39±6 vs. 36±9 ms, p<0.01), inter‐AEMD (PA lateral–PA tricuspid) (27±7 vs. 10±4 ms, p<0.01), left intra‐AEMD (PA lateral–PA septal) (18±7 vs. 10±4 ms, p<0.01), right intra‐AEMD (PA septal–PA tricuspid) (13±5 vs. 5±3 ms, p<0.01) values were higher in patients who underwent heart transplantation than in a control population. Conclusion Inter‐AEMD and intra‐AEMD were prolonged in patients who underwent heart transplantation as compared to a control population. This may explain the increased atrial fibrillation and other atrial arrhythmia incidences associated with the biatrial anastomosis heart transplantation technique and may contribute to the treatment of atrial fibrillation in this special patient group.
No takes yet. Share an insight, caveat, or question.
Bulut et al. (2016) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: