Why the study?
Is depressed tissue plasminogen activator activity associated with the development of myocardial infarction in patients with unstable angina pectoris?
Population
20 consecutive patients admitted with unstable angina pectoris
Design
Cohort
Follow-up
6 years
Authors
Loading...
Low tPA activity may mark higher MI risk in unstable angina; hypothesis-generating and should not yet change practice.
Is depressed tissue plasminogen activator activity associated with the development of myocardial infarction in patients with unstable angina pectoris?
A depressed fibrinolytic capacity due to low tissue plasminogen activator activity may be pathogenetically important for the development of myocardial infarction in patients with unstable angina.
Munkvad et al. (1990) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: