Pathological examination of ARVD and long QT syndrome cases reveals specific structural myocardial abnormalities, such as trabecular meshwork involvement in ARVD and abnormal Purkinje-like cell proliferation in long QT syndrome, which may serve as arrhythmogenic substrates.
Distinct autopsy patterns may identify arrhythmogenic substrates in ARVD and LQTS; hypothesis-generating and should not yet change practice.
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Sachio Kawai (1999) studied this question.
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