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May 28, 1996Proceedings of the National Academy of SciencesOpen Access

Recombinational repair of gaps in DNA is asymmetric in Ustilago maydis and can be explained by a migrating D-loop model.

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DFDavid O. FergusonBC Cancer AgencyWHWilliam K. HollomanCornell University

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Cite This Study

Ferguson et al. (1996) studied this question.

synapsesocial.com/papers/6a925d3bb6e367a95b6c55bahttps://doi.org/10.1073/pnas.93.11.5419
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Efficient copying of nonhomologous sequences from ectopic sites via P-element-induced gap repair.1994 · 431 citations
  2. 2Structure of REC2, a recombinational repair gene of Ustilago maydis, and its function in homologous recombination between plasmid and chromosomal sequences.1994 · 27 citations
  3. 3A general model for genetic recombination.1975 · 892 citations
  4. 4Pathways of transformation in Ustilago maydis determined by DNA conformation.1990 · 45 citations