Key result
A 55-year-old female with fulminant hepatitis exhibited transient ECG changes simulating acute myocardial infarction, likely due to catecholamine cardiomyopathy or viral myocarditis.
Case Report (n=1)
Fulminant hepatitis can be associated with transient ECG changes simulating acute myocardial infarction, potentially due to catecholamine cardiomyopathy or viral myocarditis, even in the absence of coronary artery disease.
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Fulminant hepatitis may cause transient ECG changes mimicking acute MI; single case is hypothesis-generating and should not change practice.
Serial elctrocardiograms of a 55 year old female showed a trasiently elevated ST-T wave and decreased R waves in right precordial leads suggesting anterior wall infraction during the course of fulminant hepatitis. Autopsy findings revealed marked cerebral edema, massive necrosis of the liver with lymphocytic infiltration, partial myocardial degeneration and necrosis with infiltration by lymphocytes and plasma cells. However, no significant lesions were found in the coronary arterial trees. These findings suggest that the electrocardiographic changes were due to catecholamine cardiomyopathy or viral myocarditis.
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Yoshida et al. (1979) conducted a case report in Fulminant hepatitis with ECG changes simulating acute myocardial infarction (n=1). Fulminant hepatitis was evaluated. A 55-year-old female with fulminant hepatitis exhibited transient ECG changes simulating acute myocardial infarction, likely due to catecholamine cardiomyopathy or viral myocarditis.
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