Key result
Sendai virus accessory C proteins inhibit copy-back RNA replication by increasing the selectivity of the replicating complex for promoter cis-acting elements.
The Sendai virus accessory C proteins appear to inhibit replication by increasing the selectivity of the replicating complex for promoter cis-acting elements.
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No immediate clinical translation from this animal model; leaves open broader applicability to human viral pathogens.
Tapparel et al. (1997) studied Sendai virus infection. Sendai virus accessory C proteins vs. Absence of C proteins was evaluated on Inhibition of copy-back RNA replication. Sendai virus accessory C proteins inhibit copy-back RNA replication by increasing the selectivity of the replicating complex for promoter cis-acting elements.
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