Key result
Cocaine exposure induces acute and chronic cardiovascular toxicity, including increased blood pressure, coronary vasoconstriction, myocardial necrosis, and ventricular hypertrophy.
Why the study?
What are the mechanisms and manifestations of cocaine-induced cardiovascular toxicity?
Design
Review
Authors
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Cocaine use warrants clinical vigilance in acute cardiac presentations; leaves open targeted prevention strategies.
What are the mechanisms and manifestations of cocaine-induced cardiovascular toxicity?
This review highlights the multifaceted mechanisms of cocaine cardiovascular toxicity, including sympathetic overstimulation and direct myocardial depression, which can lead to lethal cardiovascular events.
Charles W. Schindler (1996) conducted a review in Cardiovascular toxicity. Cocaine was evaluated. Cocaine exposure induces acute and chronic cardiovascular toxicity, including increased blood pressure, coronary vasoconstriction, myocardial necrosis, and ventricular hypertrophy.
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