Key result
The uric acid/albumin ratio was an independent predictor for coronary slow flow and was superior to other inflammation-based markers in detecting it (p < 0.05 for all).
Why the study?
The effect of the uric acid/albumin ratio on coronary slow flow and its relationship with other inflammation-based markers were not clearly known.
Does the uric acid/albumin ratio (UAR) predict coronary slow flow better than other inflammation-based markers?
Case-Control (n=252)
Does the uric acid/albumin ratio (UAR) predict coronary slow flow better than other inflammation-based markers?
Answers are grounded in this paper and cite their sources.
The uric acid/albumin ratio is an independent predictor of coronary slow flow and outperforms other inflammation-based markers in its detection.
Background: Many inflammation-based markers (IBMs) have been shown to be closely related to coronary slow flow (CSF), but the effect of the uric acid/albumin ratio (UAR) on CSF and its relationship with other IBMs are not clearly known. In this study, we aimed to compare the effects of UAR and other IBMs on CSF. Methods: After the exclusion criteria, 126 patients with CSF detected on coronary angiography and 126 subjects with normal coronary flow as the control group were included in the study. Results: UAR was determined as an independent predictor for CSF. In addition, the UAR was superior to other IBMs in detecting CSF (p < 0.05 for all). Conclusion: This study is the first to investigate the effect of UAR on CSF in comparison with other IBMs.
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Toprak et al. (2024) conducted a case-control in Coronary slow flow (n=252). Uric acid/albumin ratio (UAR) vs. Other inflammation-based markers was evaluated on Detection of coronary slow flow (p=<0.05). The uric acid/albumin ratio was an independent predictor for coronary slow flow and was superior to other inflammation-based markers in detecting it (p < 0.05 for all).
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