Key result
Left ventricular asynergy in patients with acute subarachnoid hemorrhage was associated with a significantly higher incidence of pulmonary edema (42% vs 12%) and elevated plasma catecholamine levels.
Why the study?
What are the clinical characteristics, hemodynamics, and catecholamine profiles of patients who develop left ventricular asynergy during the acute phase of subarachnoid hemorrhage?
Observational (n=494)
No
What are the clinical characteristics, hemodynamics, and catecholamine profiles of patients who develop left ventricular asynergy during the acute phase of subarachnoid hemorrhage?
Left ventricular asynergy in acute subarachnoid hemorrhage is a reversible phenomenon associated with elevated catecholamines, myocardial necrosis, and a high risk of pulmonary edema.
Answers are grounded in this paper and cite their sources.
LV asynergy may flag higher pulmonary edema risk in acute SAH; leaves open whether catecholamine modulation alters outcomes.
No abstract yet. The journal hasn't published one we can show.
No takes yet. Share an insight, caveat, or question.
Masuda et al. (1994) conducted an observational in Subarachnoid hemorrhage (n=494). Left ventricular asynergy vs. No left ventricular asynergy was evaluated on Incidence of pulmonary edema (p=<0.001). Left ventricular asynergy in patients with acute subarachnoid hemorrhage was associated with a significantly higher incidence of pulmonary edema (42% vs 12%) and elevated plasma catecholamine levels.
Synapse has enriched 10 related papers. Consider them for comparative context: