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September 1, 2000Molecular and Cellular BiologyOpen Access

Potentiation of GATA-2 Activity through Interactions with the Promyelocytic Leukemia Protein (PML) and the t(15;17)-Generated PML-Retinoic Acid Receptor α Oncoprotein

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Authors

STShinobu TsuzukiAichi Medical UniversityMTMasayuki TowatariNagoya UniversityHSHidehiko SaitoSeirei Hamamatsu General Hospital

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Cite This Study

Tsuzuki et al. (2000) studied this question.

synapsesocial.com/papers/6a93dd080533089f4392c29bhttps://doi.org/10.1128/mcb.20.17.6276-6286.2000
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Deconstructing a Disease: RAR, Its Fusion Partners, and Their Roles in the Pathogenesis of Acute Promyelocytic Leukemia1999 · 1,033 citations
  2. 2Murine and human T-lymphocyte GATA-3 factors mediate transcription through a cis-regulatory element within the human T-cell receptor delta gene enhancer.1991 · 297 citations
  3. 3PML, a growth suppressor disrupted in acute promyelocytic leukemia.1994 · 293 citations
  4. 4Functional analysis and in vivo footprinting implicate the erythroid transcription factor GATA-1 as a positive regulator of its own promoter.1991 · 332 citations
  5. 5PML protein expression in hematopoietic and acute promyelocytic leukemia cells1993 · 238 citations