Key result
Visual loss after heart surgery is a rare complication provoked by optic ischemia and infarction due to microembolism and/or hypoperfusion during on-pump cardiac procedures.
Maintaining stable hemodynamic parameters during cardiovascular surgery is crucial to prevent rare but devastating visual impairment caused by microembolism or hypoperfusion.
May warrant hemodynamic vigilance in on-pump surgery; leaves open prospective prevention trials.
The aim of this article was to provide a comprehensive review of current knowledge regarding ocular hemodynamic alterations affecting the retinal neuroglial cells and optic nerve head (ONH) function during cardiac surgery. Literature indicates that visual loss after heart surgery is a rare but devastating complication provoked by two main causes of optic ischaemia and infarction during on-pump cardiac procedures: microembolism and/or hypoperfusion. Retinal ischaemia and ischaemic optic neuropathy are two possible major consequences of extracorporeal circulation in cardiac surgery. The hemodynamic modifications within the vascular beds of retina and ONH during cardiovascular operations have been incompletely studied. Consequently, it is of great interest to investigate the hemodynamic changes during cardiopulmonary bypass within the choroidal, retinal and optic nerve microcirculations as well as other potential causes of vaso-occlusion. Maintaining stable hemodynamic parameters during cardiovascular surgery seems to be the key to prevent visual impairment.
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Nenekidis et al. (2011) conducted a review in Vision impairment during cardiac surgery. Extracorporeal circulation during cardiac surgery was evaluated. Visual loss after heart surgery is a rare complication provoked by optic ischemia and infarction due to microembolism and/or hypoperfusion during on-pump cardiac procedures.
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