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July 7, 2004Journal of NeuroscienceOpen Access

β-Amyloid-Mediated Inhibition of NMDA Receptor-Dependent Long-Term Potentiation Induction Involves Activation of Microglia and Stimulation of Inducible Nitric Oxide Synthase and Superoxide

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Authors

QWQinwen WangNingbo UniversityMRMichael J. RowanTrinity College DublinRARoger AnwylTrinity College

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Wang et al. (2004) studied this question.

synapsesocial.com/papers/6a947047fde2ed39e1a0d882https://doi.org/10.1523/jneurosci.0233-04.2004
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Blockade of p38 Mitogen-activated Protein Kinase Pathway Inhibits Inducible Nitric-oxide Synthase Expression in Mouse Astrocytes1997 · 302 citations
  2. 2Two forms of long-term potentiation in area CA1 activate different signal transduction cascades1996 · 189 citations
  3. 3A Cell Surface Receptor Complex for Fibrillar β-Amyloid Mediates Microglial Activation2003 · 503 citations
  4. 4β-Amyloid Stimulation of Inducible Nitric-oxide Synthase in Astrocytes Is Interleukin-1β- and Tumor Necrosis Factor-α (TNFα)-dependent, and Involves a TNFα Receptor-associated Factor- and NFκB-inducing Kinase-dependent Signaling Mechanism2000 · 363 citations
  5. 5β-Amyloid Activates the O⨪2 Forming NADPH Oxidase in Microglia, Monocytes, and Neutrophils1999 · 330 citations