Key result
Neonatal asphyxia, icterus, isoimmunization, and prematurity linked to congenital athetosis risk.
Why the study?
The relative importance of neonatal asphyxia, idiopathic icterus, ABO- and Rh-sensitization, and prematurity in the aetiology of congenital athetosis was studied.
Observational (n=214)
The study examines the etiological factors of congenital athetosis, including neonatal asphyxia, icterus, and prematurity.
Answers are grounded in this paper and cite their sources.
Hypothesis-generating for congenital athetosis etiology; prospective studies needed before implicating asphyxia, icterus, or prematurity.
In the present paper the aetiology of congenital 88 athetosis has been studied by examining the relative importance of the following factors: (1) neonatal 80o asphyxia without icterus, (2) neonatal icterus of 72 unknown origin, (3) neonatal icterus due to ABO- 64 and Rh-sensitization, and (4) prematurity.56-Material and Methods AGE IN YEARS * This study was supported by a PHS Research Grant B-2408 from The National Institute of Neurological Diseases and Blindness, FIG.2.-Age in years at last examination of 173 cases of athetosis Bethesda, U.S.A., and by The Danish Cerebral Palsy Fund.(white area).Age at death of 41 cases of athetosis (hatched area).
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P Plum (1965) conducted an observational in Congenital athetosis (n=214). Neonatal asphyxia, idiopathic icterus, ABO and Rh-sensitization, and prematurity was evaluated on Aetiology of congenital athetosis. This observational study evaluated the aetiological role of neonatal asphyxia, icterus of unknown origin, ABO/Rh-sensitization, and prematurity in 214 cases of congenital athetosis.
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