December 1, 2002Journal of Medical GeneticsOpen Access

PRKAR1A, one of the Carney complex genes, and its locus (17q22-24) are rarely altered in pituitary tumours outside the Carney complex: Figure 1

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Population

Large collection of sporadic pituitary adenomas (SPA) from the USA, UK, Japan, and France

Design

Other

Authors

FSFabiano SandriniNational Institutes of Health

Discussion

Member takes

Implication

May implicate PRKAR1A in sporadic pituitary adenomas; leaves open clinical genetic testing beyond Carney complex.

Structured PICO

Population

Large collection of sporadic pituitary adenomas (SPA) from the USA, UK, Japan, and France

Outcome

Loss of heterozygosity (LOH) of the 17q22-24 PRKAR1A locussurrogate

The study investigates the frequency of PRKAR1A gene alterations and 17q22-24 locus loss of heterozygosity in sporadic pituitary adenomas outside of the Carney complex.

Cite This Study

Fabiano Sandrini (2002) studied this question.

synapsesocial.com/papers/6a95406da15fb1352cb2e96a https://doi.org/10.1136/jmg.39.12.e78

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PRKAR1A, one of the Carney complex genes, and its locus (17q22-24) are rarely altered in pituitary tumours outside the Carney complex: Figure 1

Discussion

Member takes

Implication

Structured PICO

Cite This Study

PRKAR1A, one of the Carney complex genes, and its locus (17q22-24) are rarely altered in pituitary tumours outside the Carney complex: Figure 1

Discussion

Member takes

Implication

Structured PICO

Cite This Study

Also Consider

Also Consider