Key result
A 15-year-old female with repeated aborted sudden cardiac death, anomalous RCA origin, and congenital LQTS remained free from ventricular arrhythmias for over 6 months after beta-blocker therapy.
Case Report (n=1)
The coexistence of an anomalous origin of the right coronary artery and congenital long QT syndrome can interact to induce ventricular arrhythmias, highlighting the importance of evaluating for QT prolongation even when a structural coronary anomaly is identified.
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Beta-blockers may suppress arrhythmias in combined anomalous coronary origin and LQTS; hypothesis-generating and requires confirmation beyond this case.
A 15-year-old female with a prior history of aborted cardiac death and surgical correction of anomalous origin of the right coronary artery (RCA) presented with polymorphic ventricular tachycardia. Her electrocardiogram after defibrillation was suggestive of congenital long QT syndrome (LQTS). The patient was treated with a β-blocker and remained free from ventricular arrhythmia during the follow-up of more than 6 months. Here, we present the case of a young female with repeated aborted cardiac death accompanied by anomalous origin of the RCA and congenital LQTS for the first time.
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Park et al. (2013) conducted a case report in Repeated aborted sudden cardiac death, anomalous origin of the right coronary artery, and congenital long QT syndrome (n=1). Beta-blocker (atenolol) was evaluated on Ventricular arrhythmia recurrence. A 15-year-old female with repeated aborted sudden cardiac death, anomalous RCA origin, and congenital LQTS remained free from ventricular arrhythmias for over 6 months after beta-blocker therapy.
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