Key result
Epicardial adipose tissue is associated with thin cap fibroatheromas, with imaging modalities like optical coherence tomography and multislice computed tomography playing a role in their assessment.
This review highlights the pathophysiological association between epicardial adipose tissue and vulnerable coronary plaques (thin cap fibroatheromas), and discusses the utility of OCT and MSCT in their assessment.
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May aid noninvasive identification of vulnerable plaques; leaves open causal role and therapeutic targeting of epicardial adipose tissue.
Thin cap fibroatheromas (TCFAs) are thought to be the most common underlying substrate in patients suffering acute coronary thrombotic events. Recently, an interesting association between TCFAs and a particular depot of visceral fat called epicardial adipose tissue (EAT) has been suggested. In this article, we discuss some basic and clinical aspects of this association and then briefly review some of the pathophysiological characteristics attributed to EAT that explain why this particular depot of fat has been attracting the attention of the cardiological scientific community in recent years. Finally we discuss the value of optical coherence tomography in the diagnosis of TCFAs and the role of multislice computed tomography to assess EAT.
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Mauro Echavarría‐Pinto (2013) conducted a review in Thin cap fibroatheromas and acute coronary thrombotic events. Epicardial adipose tissue (EAT) was evaluated. Epicardial adipose tissue is associated with thin cap fibroatheromas, with imaging modalities like optical coherence tomography and multislice computed tomography playing a role in their assessment.
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