Post-traumatic syringomyelia was previously thought to be an infrequent but serious sequel to spinal cord injury. Clinical and CT studies have shown an incidence of between 1% and 5%, but more recently MRI has suggested an incidence of up to 22%. Twenty spinal cords have been examined after death from two days to 43 years after injury. Four had syrinxes, 20% of the series, approaching the incidence found by MRI. The acute and chronic pathological changes after trauma are described. Post-traumatic syringomyelia seems to develop from cores of necrotic tissue (myelomalacic cores) rather than lysis of haematoma. The mechanism of extension of syrinxes remains unexplained.
No takes yet. Share an insight, caveat, or question.
Squier et al. (1994) studied this question.
Synapse has enriched 5 related papers. Consider them for comparative context: