Suckling mouse cataract agent (SMCA) inoculated intracerebrally in newborn rats results in persistence of infectious agent in the brain over a considerable period of time concomitant with hydrocephalus. The means by which the SMCA infection causes hydrocephalus remains obscure; a functional block in the cerebrospinal fluid pathways is postulated.
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Elizan et al. (1972) studied this question.