Key result
Epicardial fat acts as an active endocrine organ that can dramatically affect vessels and is a predictor for essential hypertension and early vascular damage.
Why the study?
Does epicardial fat predict essential hypertension and early vascular damage in patients with metabolic syndrome and non-alcoholic fatty liver disease?
Does epicardial fat predict essential hypertension and early vascular damage in patients with metabolic syndrome and non-alcoholic fatty liver disease?
Epicardial fat is highlighted as an active endocrine organ and a potential predictor for essential hypertension and early vascular damage in patients with metabolic syndrome and NAFLD.
Epicardial fat may inform vascular risk assessment in metabolic syndrome with NAFLD; leaves open incremental value and need for prospective validation.
Increased visceral adiposity is a new marker of metabolic syndrome, early vascular damage, non-alcoholic fatty liver disease and also is associated with essential hypertension. Epicardial adipose tissue is a very important factor in the pathogenesis of coronary atherosclerosis. Hypertension is one of the most important risk factor for the development of coronary heart disease, stroke, congestive heart failure, and end-stage renal disease. Epicardial fat is an active endocrine organ producing a wide range of active molecules, which can affect vessels dramatically.
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Drapkina et al. (2014) conducted a review in Metabolic syndrome and non-alcoholic fatty liver disease. Epicardial fat was evaluated. Epicardial fat acts as an active endocrine organ that can dramatically affect vessels and is a predictor for essential hypertension and early vascular damage.
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