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April 1, 1997Endocrinology

1,25-Dihydroxyvitamin D3 and 9-cis-Retinoic Acid Act Synergistically to Inhibit the Growth of LNCaP Prostate Cells and Cause Accumulation of Cells in G1*

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Authors

SBSarah E. BluttBaylor College of MedicineEAElizabeth A. AllegrettoLigand Pharmaceuticals (United States)JPJ. Wesley PikeUniversity of North Carolina at Chapel Hill

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Cite This Study

Blutt et al. (1997) studied this question.

synapsesocial.com/papers/6a9a3d984e49e699219e6579https://doi.org/10.1210/endo.138.4.5063
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Actions of vitamin D3, analogs on human prostate cancer cell lines: comparison with 1,25-dihydroxyvitamin D3.1995 · 203 citations
  2. 2Simian virus 40-, but not human papillomavirus-, transformation of prostatic epithelial cells results in loss of growth-inhibition by 1,25-dihydroxyvitamin D-31996 · 18 citations
  3. 3Stable expression of the nuclear vitamin D receptor in the human prostatic carcinoma cell line JCA-1: evidence that the antiproliferative effects of 1 alpha, 25-dihydroxyvitamin D3 are mediated exclusively through the genomic signaling pathway.1996 · 116 citations
  4. 4VITAMIN D DERIVATIVES IN COMBINATION WITH 9- cis RETINOIC ACID PROMOTE ACTIVE CELL DEATH IN BREAST CANCER CELLS1995 · 89 citations
  5. 5Retinoic Acid Receptors and Cellular Retinoid Binding Proteins: Complex Interplay in Retinoid Signaling*1994 · 514 citations