Key result
High blood viscosity in a fluid-structure interaction model reduced velocity amplitude and enhanced pressure level without changing the time delay of pulse wave propagation.
Why the study?
Does high blood viscosity affect pulse wave propagation compared to normal viscosity in a fluid-structure interaction model?
Does high blood viscosity affect pulse wave propagation compared to normal viscosity in a fluid-structure interaction model?
Computational modeling demonstrates that high blood viscosity alters pulse wave hemodynamics by reducing velocity amplitude and increasing pressure, providing physical insight into its role in cardiovascular disease.
High viscosity alters modeled hemodynamics without affecting propagation delay; leaves open clinical relevance for pulse wave assessment.
High blood viscosity often causes cardiovascular diseases, such as hypertension, atherosclerosis and thrombosis. It is proven that blood viscosity plays an important role in cardiovascular functions. In this paper, pulse wave characteristics with normal and high blood viscosities are presented in detail to evaluate how blood viscosity affects pulse wave propagation. A fluid-structure interaction is employed to solve for pulse wave characteristics. The results show that increased blood viscosity does not change the time delay of wave propagation. However, high viscosity reduces the velocity amplitude, while it enhances the pressure level. This study provides physical insight for evaluating blood viscosity leading potentially to pulse wave changes.
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He et al. (2014) studied Pulse wave propagation. High blood viscosity vs. Normal blood viscosity was evaluated on Pulse wave characteristics (time delay, velocity amplitude, pressure level). High blood viscosity in a fluid-structure interaction model reduced velocity amplitude and enhanced pressure level without changing the time delay of pulse wave propagation.
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