Key result
Severe COVID-19 pneumonia can be associated with the development of stress cardiomyopathy (Takotsubo syndrome), presenting with typical EKG and echocardiographic changes.
Why the study?
Patients with COVID-19 pneumonia may develop stress cardiomyopathy (Takotsubo syndrome) at different stages and with varying degrees of left ventricular dysfunction.
Case Report (n=3)
No
COVID-19 infection may trigger stress cardiomyopathy (Takotsubo syndrome), highlighting the need to monitor for cardiac involvement and consider postponing invasive coronary angiography until pneumonia resolution to prevent virus spread.
TTS may complicate COVID-19 pneumonia with variable outcomes; case series leaves open optimal diagnostic timing amid infection risks.
Patients affected by COVID-19 pneumonia may develop stress cardiomyopathy, also known as Takotsubo syndrome (TTS), at different stages during the disease and with different degrees of left ventricular dysfunction. We describe three cases of TTS in COVID-19-positive patients with different clinical presentations and outcomes. One of them died, while in the other two coronary angiography confirmed the diagnosis but was postponed until after pneumonia resolution because of the risk of virus spread. LEARNING POINTS: An association between COVID-19 and cardiac involvement is highlighted.The incidence of Takotsubo syndrome has increased during this pandemic, possibly because it is caused by acute stress.
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Pasqualetto et al. (2020) conducted a case report in COVID-19 and stress cardiomyopathy (Takotsubo syndrome) (n=3). COVID-19 infection was evaluated. Severe COVID-19 pneumonia can be associated with the development of stress cardiomyopathy (Takotsubo syndrome), presenting with typical EKG and echocardiographic changes.
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