Key result
DEK autoantibodies and protein in the synovial fluid of JIA patients may contribute to joint inflammation by generating immune complexes, a process enhanced by DEK acetylation.
Authors
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Should not yet change JIA practice; leaves open whether DEK autoantibodies or acetylation drive inflammation.
Mor‐Vaknin et al. (2010) studied juvenile idiopathic arthritis (JIA). DEK protein and autoantibodies was evaluated on Characterization of DEK autoantibodies and posttranslational modification of DEK. DEK autoantibodies and protein in the synovial fluid of JIA patients may contribute to joint inflammation by generating immune complexes, a process enhanced by DEK acetylation.
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